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These tools ensure that key diagnostic features are systematically evaluated rather than left to provider memory or initiative (EHP-30 Study Group, 2001)

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However, observed barrier defects and associated diarrhea even in these mice did not result in changes in claudin-1, -2, -3, and -4 expression and cellular distributions, but rather correlated with the rearrangement of cellular distribution of occludin, and to some extent with JAM proteins