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These authors also confirmed that the intact disulphide form of these ETP moieties was essential for Grx1 inactivation and that prior reduction of sporidesmin, using glutathione, prevented subsequent Grx1 inactivation

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While the extent of arterial labeling is limited in these short-term genetic fate tracking experiments, the results indicate that Esm1-CreERT2 -labeled cells are incorporated into mesenteric arteries but not into nearby veins or lymphatic vessels (Fig

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