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However in our opinion, the low probability of MS coincidence with another rare disease, together with the animal studies where MS is never a consideration, makes it more likely that these human examples align with an inside-out model (Figure 2), where a primary biochemical derangement of myelin, coupled with an immune predisposition, culminated in a MS-like phenotype

The patient's eyes were open, ears were unoccluded, and ambient noise was kept to a minimum during the study

Debatri Datta April 10, 2024 Written By Dr

If our hypothesis is confirmed (i.e., some of these host cleavage sites prove to be functional), that leaves us with an interesting questionwhat evolutionary advantages are driving some viruses to go so far as to degrade or block the synthesis of GSH and specific host selenoproteins