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Damaged AECs release pro-inflammatory and pro-fibrotic mediators, including interleukin (IL) -1 and tumor necrosis factor (TNF) -, which promote fibroblast recruitment, inflammatory cell activation (e.g., neutrophils, Th17, gdT cells), and myofibroblast differentiation, ultimately leading to ECM accumulation and fibrotic tissue remodeling [20, 29, 33, 34]

Expression profiling using the microarray data provided significant evidence of their participation in response to various abiotic stress conditions

Side Effects Comparison# CagriSema Side Effects# GI adverse events occurred in 79.6% of CagriSema recipients versus 39.9% on placebo in REDEFINE 1

This antiviral agent, available as a 1% topical cream and a 2.5 mg/mL intralesional solution, has shown effectiveness in managing relapsing HPV lesions, including those resistant to other treatments