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showed that intracellular glutamate accumulation following cyst(e)ine depletion or system x C - inhibition is a key contributor to the induction of ferroptosis, and glutamate toxicity can be alleviated by converting excess glutamate to -glutamyl-di- or tripeptides via promiscuous enzymatic activities of glutamate-cysteine ligase catalytic subunit (GCLC) 12

That means more of the glutathione gets delivered to the bloodstream

Do you find if you write the bit out, physically write it out, that you will remember it better, too

Free Radic Biol Med 152:175185 Yang W, Mu B, You J et al (2022) Non-classical ferroptosis inhibition by a small molecule targeting PHB2