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Instead of directly targeting established inflammatory responses, this approach seeks to prevent the formation of pro-inflammatory crystals at their source by modulating crystallization pathways and the properties of crystal interfaces

Emerging evidence highlights the therapeutic potential of targeting protein palmitoylation in IBD ( Table 3 )

They can regulate appetite, metabolism, cardiovascular function, and even act as neurotransmitters, transmitting signals between nerve cells in the brain and nervous system

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