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8 Despite methyltetrahydrofolate being the physiologically active form, some studies on patients with methyltetrahydrofolate receptor C677T genotype polymorphisms (a prominent genotype in autism) suggest that folinic acid supplementation may cause higher serum folate levels than similar doses of methyltetrahydrofolate

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These findings position neutrophil ferroptosisorchestrated by GPX4as a mechanistic bridge between iron dyshomeostasis and loss of self-tolerance in SLE, offering a rationale for therapeutic strategies targeting neutrophil redox resilience or iron chelation in autoimmune disease