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Studies suggest that Epithalon may directly affect telomere length, a critical factor in cellular aging

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This complexity underscores the need for stage-specific interventions: acute mitochondrial injury may respond to antioxidants or metabolic substrates, while advanced disease may require reprogramming of cellular bioenergetics

thus, only a stratification according to these factors would ensure a reliable evaluation of biologics efficacy in reducing the likelihood of developing arthritis