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L-carnitine and its derivative ALCAR are supported by evidence as effective interventions for managing chronic neuropathic pain, with mechanisms involving nerve regeneration, neurotrophic factor modulation, and mitochondrial function enhancement

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For example, targeting receptor, ligand and downstream signaling molecules such as phosphatidylinositol 3-kinase or particularly the insulin receptor substrate adapter proteins might result in a complete blockade of insulin-like growth factor-1 receptor/insulin receptor biological functions