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In naturally aged rat ovaries, abnormal upregulation of transferrin (TF) and iron regulatory protein 2 (IRP2)-mediated transferrin receptor 1 (TfR1) leads to iron accumulation (Sze et al., 2022)

Epithalon works by upregulating telomerase activity and thus elongating telomeres, which are sections of DNA that protect the ends of chromosomes from removal during each replication cycle on account of one strand of replication fork lagging behind the other

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This approach offers a viable pathway for personalized medicine and provides new scientific insights and translational opportunities for the safe and precise treatment of inflammatory diseases