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The lack of adequate cobalamin in the body hinders the regeneration of tetrahydrofolate, which eventually leads to megaloblastic anemia due to the functional folate deficiency.[9][10] On the other hand, the methylmalonyl-CoA mutase helps to metabolize odd chain fatty acids and branch chain amino acids.[5] Cobalamin is also thought to keep the bodys level of sulfhydryl (SH) groups in reduced form

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doi: 10.3889/oamjms.2018.257

Once inside the cell, the sulfhydryl (-SH) group of the glutathione molecule acts as a potent reducing agent, directly donating an electron to unstable reactive oxygen species