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Therefore, due to the loss of inhibitory control of GDH by mutations in GLUD1 , insulin was secreted excessively under basal conditions as well as after protein meals, triggering hyperinsulinemia and hypoglycemia [15]

Skin becoming thin, fragile, or crepe-like

Importantly, PGC-1-PPAR-mediated mitochondrial biogenesis is sufficient to promote the transition from low-OXPHOS to high-OXPHOS characteristics, which is associated with better prognosis in HGSOC patients