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2 It is well known that increased estradiol synthesis promotes the proliferation of endometriotic lesions, inhibits cell apoptosis, and promotes the occurrence and development of endometriosis

Estrogen has been shown to upregulate endogenous MOTS-c expression in preclinical models, which may partly explain why metabolic deterioration accelerates in perimenopause when estrogen falls precipitously [2]

Recovery Between Sessions GH and IGF-1 play a role in: Muscle repair Connective tissue healing Bone remodeling Studies in GH-deficient populations show improved recovery markers and body composition with GH axis stimulation. While direct evidence in healthy athletes is limited, the mechanism remains relevant

Cisplatin drugs damage mature follicles and deplete the primordial follicles